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Infectious agents, especially bacteria and their components originating from the oral cavity or respiratory tract, have been suggested to contribute to inflammation in the coronary plaque, leading to rupture and the subsequent development of coronary thrombus. We aimed to measure bacterial DNA in thrombus aspirates of patients with STsegment–elevation myocardial infarction and to check for a possible association between bacteria findings and oral pathology in the same cohort.
CIRCULATIONAHA.112.001254
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